Camptocormia: Difference between revisions

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[[File:Camptocormia.png|thumb|right|A patient with camptocormia]]
[[File:Camptocormia.png|thumb|right|A patient with camptocormia]]
'''Camptocormia''' is characterised by marked flexion (greater than 45 degrees) of the thoracolumbar spine that increases during the day that can be caused by several different diseases.
'''Camptocormia''' also known as '''bent spine syndrome''' is a usually organic disorder characterised by marked flexion (greater than 45 degrees) of the thoracolumbar spine that increases during the day and completely disappears when supine.


==History and Terminology==
==Terminology==
First described by Earle in 1815 and by Brodie in 1837. The term comes from the Greek words "to bend" (kamptō) and "trunk" ("trunk").
The term comes from the Greek words "to bend" (kamptō) and "trunk" ("trunk").


==Aetiology==
==Aetiology==
Camptocormia can have a neurological or muscular origin
Camptocormia can have a neurological or muscular origin. There are a multitude of causes.
*Parkinson's disease: the most common cause (two third).
*Parkinson's disease: the most common cause (two third).
*Other parkinsonian syndromes
*Other parkinsonian syndromes
*Multisystem atrophy
*Alzheimer's disease
*Motor neuron disease
*Myaesthenia
*[[Chronic Inflammatory Demyelinating Polyneuropathy|Chronic inflammatory demyelinating polyneuropathy]]
*Dystonia
*Dystonia
*Vascular lenticular lesions
*Vascular lenticular lesions
*Muscular disorders
*Primary or secondary myopathy
*Rheumatologic disorders.
*[[Internal Disc Disruption|Lumbar disc herniation]]
*[[Osteoarthritis]]
*Spinal trauma
*Paraneoplasia
*Medication side effects (olanzapine, donepezil, valproate, systemic steroids)


==Epidemiology==
==Epidemiology==
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==Clinical Features==
==Clinical Features==
As the day goes on or during walking there is a progressive marked flexion of the thoracolumbar spine. It is relieved by sitting and lying supine. It can also be relieved volitionally by extending the trunk when the patient leans against a wall. There may be associated lateral deviation of the trunk. In two thirds of patients there is a lumbar or thoracolumbar scoliosis.<ref name=":0" />
As the day goes on or during walking there is a progressive marked flexion of the thoracolumbar spine. It is relieved by sitting and lying supine. It can also be relieved volitionally by extending the trunk when the patient leans against a wall. There may be associated lateral deviation of the trunk. In two thirds of patients there is a lumbar or thoracolumbar scoliosis.<ref name=":0" />
== Treatment ==
Treatment is usually unrewarding. Where possible treatment should be directed to the underlying cause.


==Further Reading==
==Further Reading==

Revision as of 16:05, 26 March 2022

This article is a stub.
A patient with camptocormia

Camptocormia also known as bent spine syndrome is a usually organic disorder characterised by marked flexion (greater than 45 degrees) of the thoracolumbar spine that increases during the day and completely disappears when supine.

Terminology

The term comes from the Greek words "to bend" (kamptō) and "trunk" ("trunk").

Aetiology

Camptocormia can have a neurological or muscular origin. There are a multitude of causes.

Epidemiology

In those with Parkinson's disease and camptocormia there is a male predominance, older age, longer duration of disease, and autonomic symptoms.[1]

Clinical Features

As the day goes on or during walking there is a progressive marked flexion of the thoracolumbar spine. It is relieved by sitting and lying supine. It can also be relieved volitionally by extending the trunk when the patient leans against a wall. There may be associated lateral deviation of the trunk. In two thirds of patients there is a lumbar or thoracolumbar scoliosis.[1]

Treatment

Treatment is usually unrewarding. Where possible treatment should be directed to the underlying cause.

Further Reading

References

  1. ā†‘ 1.0 1.1 Benatru, I.; Vaugoyeau, M.; Azulay, J.-P. (2008-12). "Postural disorders in Parkinson's disease". Neurophysiologie Clinique = Clinical Neurophysiology. 38 (6): 459ā€“465. doi:10.1016/j.neucli.2008.07.006. ISSN 0987-7053. PMID 19026965. Check date values in: |date= (help)

Literature Review