Camptocormia
Camptocormia also known as bent spine syndrome is a usually organic disorder characterised by marked flexion (greater than 45 degrees) of the thoracolumbar spine that increases during the day and completely disappears when supine.
Terminology
The term comes from the Greek words "to bend" (kamptÅ) and "trunk" ("trunk").
Aetiology
Camptocormia can have a neurological or muscular origin. There are a multitude of causes.
- Parkinson's disease: the most common cause (two third).
- Other parkinsonian syndromes
- Multisystem atrophy
- Alzheimer's disease
- Motor neuron disease
- Myaesthenia
- Chronic inflammatory demyelinating polyneuropathy
- Dystonia
- Vascular lenticular lesions
- Primary or secondary myopathy
- Lumbar disc herniation
- Osteoarthritis
- Spinal trauma
- Paraneoplasia
- Medication side effects (olanzapine, donepezil, valproate, systemic steroids)
Epidemiology
In those with Parkinson's disease and camptocormia there is a male predominance, older age, longer duration of disease, and autonomic symptoms.[1]
Clinical Features
As the day goes on or during walking there is a progressive marked flexion of the thoracolumbar spine. It is relieved by sitting and lying supine. It can also be relieved volitionally by extending the trunk when the patient leans against a wall. There may be associated lateral deviation of the trunk. In two thirds of patients there is a lumbar or thoracolumbar scoliosis.[1]
Treatment
Treatment is usually unrewarding. Where possible treatment should be directed to the underlying cause.
Further Reading
References
- ā 1.0 1.1 Benatru, I.; Vaugoyeau, M.; Azulay, J.-P. (2008-12). "Postural disorders in Parkinson's disease". Neurophysiologie Clinique = Clinical Neurophysiology. 38 (6): 459ā465. doi:10.1016/j.neucli.2008.07.006. ISSN 0987-7053. PMID 19026965. Check date values in:
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Literature Review
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